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| Dot/Icm T4SS |
 |
Highly homologous to the L. pneumophila Dot/Icm system. Facilitates the translocation of many bacterial effector proteins across the bacterial and vacuole membranes to enter the host cytoplasm where the effector proteins mediate their specific activities to manipulate a variety of host cell processes. ... |
|
Dot/Icm
(Defect in organella trafficking/intracellular multiplication) |
|
14 genes share detectable homology to the tra/trb genes of col1b-P9 plasmid, a member of the IncI class of conjugal plasmids. The second type IV secretion apparatus lvh (Legionella vir homologs) system is poorly characterized. Its 11 genes are located on a genomic-island-like region. It might play a role in the efficiency of host cell infection by L. pneumophila grown at lower temperatures. However, the specific substrates of this system have not yet been identified. ... |
|
| Dot/Icm T4SS |
|
Highly homologous to the L. pneumophila Dot/Icm system. Facilitates the translocation of many bacterial effector proteins across the bacterial and vacuole membranes to enter the host cytoplasm where the effector proteins mediate their specific activities to manipulate a variety of host cell processes. ... |
|
| Dot/Icm T4SS |
|
Highly homologous to the L. pneumophila Dot/Icm system. Facilitates the translocation of many bacterial effector proteins across the bacterial and vacuole membranes to enter the host cytoplasm where the effector proteins mediate their specific activities to manipulate a variety of host cell processes. ... |
|
| Dot/Icm T4SS |
|
Highly homologous to the L. pneumophila Dot/Icm system. Facilitates the translocation of many bacterial effector proteins across the bacterial and vacuole membranes to enter the host cytoplasm where the effector proteins mediate their specific activities to manipulate a variety of host cell processes. ... |
|
| Dot/Icm T4SS |
|
Highly homologous to the L. pneumophila Dot/Icm system. Facilitates the translocation of many bacterial effector proteins across the bacterial and vacuole membranes to enter the host cytoplasm where the effector proteins mediate their specific activities to manipulate a variety of host cell processes. ... |
|
Dot/Icm
(Defect in organella trafficking/intracellular multiplication) |
|
14 genes share detectable homology to the tra/trb genes of col1b-P9 plasmid, a member of the IncI class of conjugal plasmids. The second type IV secretion apparatus lvh (Legionella vir homologs) system is poorly characterized. Its 11 genes are located on a genomic-island-like region. It might play a role in the efficiency of host cell infection by L. pneumophila grown at lower temperatures. However, the specific substrates of this system have not yet been identified. ... |
|
Dot/Icm
(Defect in organella trafficking/intracellular multiplication) |
|
14 genes share detectable homology to the tra/trb genes of col1b-P9 plasmid, a member of the IncI class of conjugal plasmids. The second type IV secretion apparatus lvh (Legionella vir homologs) system is poorly characterized. Its 11 genes are located on a genomic-island-like region. It might play a role in the efficiency of host cell infection by L. pneumophila grown at lower temperatures. However, the specific substrates of this system have not yet been identified. ... |
|
Dot/Icm
(Defect in organella trafficking/intracellular multiplication) |
|
14 genes share detectable homology to the tra/trb genes of col1b-P9 plasmid, a member of the IncI class of conjugal plasmids. The second type IV secretion apparatus lvh (Legionella vir homologs) system is poorly characterized. Its 11 genes are located on a genomic-island-like region. It might play a role in the efficiency of host cell infection by L. pneumophila grown at lower temperatures. However, the specific substrates of this system have not yet been identified. ... |
|
Dot/Icm
(Defect in organella trafficking/intracellular multiplication) |
|
14 genes share detectable homology to the tra/trb genes of col1b-P9 plasmid, a member of the IncI class of conjugal plasmids. The second type IV secretion apparatus lvh (Legionella vir homologs) system is poorly characterized. Its 11 genes are located on a genomic-island-like region. It might play a role in the efficiency of host cell infection by L. pneumophila grown at lower temperatures. However, the specific substrates of this system have not yet been identified. ... |
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